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幽门螺旋杆菌及其致病机理的研究进展

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licobacterpyloriHeatShockProtein60MediatesInterleukin-6ProductionbyMacrophagesviaaToll-likeReceptor(TLR)-2-,TlT-4-,andMyeloidDifferentiationFactor88-independentMechanism[J].JBC,2004,279(1):245-250.[80]GoertAP,McGeeDJ,AkhtarM,etal.ells:Astrategyforbacterialsurvival[J].PNAS,2001,98(24):13844-13849.[81]ChengY,ChaturvediR,AsimM,etal.Helicobacterpylori-inducedMacrophageApoptosisRequieresActivationofOrnithineDecarboxylasebyc-Myc[J].TheJournalofBiologicalChemistry,2005,280(23):22492-22496.[82]GobertAP,ChengY,WangJY,etal.HelicobacterpyloriInducesMacrophageApoptosisbyActivationofArginaseII[J].JImmunol,2002,168:4692-4700.[83]赵亚刚,张小瑞,吴炜景等.诱导性一氧化氮合酶在幽门螺杆菌与胃癌关系中的研究进展[J].实用医学杂志,2009,25(7):1166-1167.[84]KonturekPC,KonturekSJ,BrzozowskiT.ancerogenesis[J].JournalofPhysiologyandPharmacology,2009,60(3):3-21.

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